Carbolic = clinometric drug that binds and activates the acetylene’s receptor. It is a choleric agonies used for treating glaucoma or during ophthalmic surgery.
Cooling cerebrate Positively charged quaternary ammonium compound Not well absorbed In GAIT tract and does not cross the BOB Not easily metabolites by cholinesterase 2-5 min onset of action Duration of action 4-8 hours (topical), 24 hours for intraocular administration Parasympathetic’s that stimulates both musicians and nicotine receptors Methodological = Synthetic cooling ester, acts as a non-selective musicians receptor consist in the parasympathetic nervous system.
Used to diagnose bronchial hyperactivity (asthma and chronic pulmonary obstructive disease). Adverse effects – brickyard and hypertension as it is a clinometric. Little effect on nicotine receptors Charged quaternary amine structure – essential for activity Does not cross BOB Not easily broken down by psychotherapeutics (broken down at slow rate) however the ester makes It susceptible to AChE Beta-methyl group – selectivity towards musicians receptors compared to nicotine receptors Benevolence = Strong nicotine-like properties and weak mescaline-like repositories.
Contains a benzene ester grouping (like many local anesthetics) Exhumations = nicotine acetylene’s receptor agonies – induce muscle relaxation and short term paralysis. Depilatories neuromuscular blocker, acting on nicotine receptors to result in persistent deportations of the motor end plate. It is degraded by butyrylcholinesterase (plasma cholinesterase). Hydrolysis by Beech is much slower than that of ACh by AChE. INHIBITORS Physiognomies: (serine) parasympathetic’s alkaloid, reversible cholinesterase inhibitor.
Acts by interfering with metabolism of ACh. It is a covalent inhibitor reversible – bond hydrolysis and released) of AChE. It indirectly stimulates both nicotine and musicians acetylene’s receptors. Used to treat glaucoma, Listener’s disease and delayed gastric emptying. It is an amine and can cross BOB.
Main function is to prevent the hydrolysis of acetylene’s by psychotherapist’s at the transmitted sites of acetylene’s. Inhibition of AChE enhances effect of ACh thus useful for the treatment of choleric disorders and anesthesia gravies.
Moistening = parasympathetic’s – reversible AChE Inhibitor. Interferes with the breakdown of ACh – stimulates nicotine and muscularly receptors.
Unlike physiologists, engrossment has a quaternary nitrogen, thus Is more polar and does not enter the CONS. Effect on skeletal muscle Is greater than physiologists. Moderate duration of action, like 2-4 hours. Binds to the anionic and esoteric site of AChE.
Drug posthypnotic membrane receptors. Used to improve muscle tone in people with anesthesia gravies and in antithesis to reverse effects of noon-depilatories muscle relaxants.
Dropping readily reversible AChE inhibitor. Prevents breakdown of ACh by competitively inhibiting ACHe mainly at the neuromuscular Junction.
Used to preferential anesthesia gravies from choleric crisis and Lambert-Eaton anesthetic syndrome. Dropping reduces the muscle weakness prolonging ACh presence in synaptic cleft. Anesthesia gravies – the antibodies attack/block/destroy the nicotine acetylene’s receptors so dropping reduces muscle weakness.
In choleric crisis (person has too much stimulation) dropping makes muscle weakness worse by inducing a depilatories block. ELMS – neuron is unable to release enough ACh for normal muscle function due to authentication PIG type calcium channel necessary for acetylene’s release so there is insufficient calcium ion influx thus educed ACh release.
Thus not much increase in muscle strength after dropping injection, if any. Immolation = orchestrating parasympathetic’s – irreversible binding to cholinesterase.
Used as an insecticide of low human toxicity. Malone – a phosphorescent – breakdown product of immolation and is much more toxic. Ester. Atropine Atropine alkaloid extracted from deadly nightshade (troop belladonna) etc.
Secondary metabolite of these plants. Drug – wide variety of effects. Generally, atropine counters “rest and digest” activity regulated by parasympathetic nervous yester BECAUSE atropine is a competitive antagonist of the musicians acetylene receptors – (ACh is main neurotransmitter used by the parasympathetic nervous system.
Atropine dilates pupils, increases heart rate and reduces salivation and other secretions. Used to treat orchestrating poisoning – not an antidote, but blocks action of ACh at musicians receptors – also serves at treatment for poisoning by insecticides, nerve gases like tab, saris, somas and VS..
Racemes mixture Pharmacological effects due to binding to musicians acetylene’s receptors – antisubmarine agent